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Clinical Trial - Testosterone Levels after the Consumption of 1:200 Tongkat Ali Extract from Indonesia


If the testosterone levels of adolescent malesare raised with potent 1:200 Tongkat Ali Extract from Indonesia, this can be dangerous for human communities. Younger men may find it hard to manage extraordinary sexual agitation, especially after larger dosages like those for older men. Therefore, the minimum age of male trial participants should be 25 years. There is no age limit for female trial participants.

Trial participants should not have been using genuine Tongkat Ali products for at least one year. Before trial participants start to consume 1:200 Tongkat Ali Extract from Indonesia, they should have their blood tested for testosterone A (bound to Sex Hormone Binding Globulin) and for testosterone B (not bound to Sex Hormone Binding Globuline).

Test results should be sent as email attachments to Tongkatali.org.

Low testosterone levels is an underestimated modern scourge. Low testosterone levels cause infertility of men, and disturbe sexual desire and sexual function. Low testosterone levels causes depression and a general feeling that life is not worth living. High testosterone makes optimistic. Men with high testosterone levels have an athletic shape of the body. High testosterone also creates an aura that makes men of any age look attractive to women. Men with high testosterone are often more successful in their professions. Testosterone makes intelligent and boosts the immune system.

But testosterone injections, testosterone capsules, and testosterone patches are not the solution. Many of the positive effects of testosterone are not linked to the presence of testosterone in blood plasma. Many positive effects of testosterone are linked to the fact that the body synthesizes testosterone.

In men, most testosterone is synthesized in the testes. But testosterone is also synthesized in adrenal glands and in various cell clusters along the axis of the hypothalamus, the pituitary gland, and the gonads. Along the axis of the hypothalamus, the pituitary gland, and the gonads, testosterone interacts with other hormones and with neurotransmitters like dopamine. Sexual phantasies and sexual satisfaction are related to the intereaction of the synthesis of both testosterone and dopamine in the preoptic area of the hypothalamus. A lack of sexual excitement and sexual satisfaction resulting from a hypothalamus that is out of tune cannot be medicated with testosterone injections, testosterone capsules, or testosterone patches.

Testosterone injections, testosterone capsules, and testosterone patches are effective in sports doping and bodybuilding. But for sexual parameters, all three will cause more harm than benefit.

1:200 Tongkat Ali Extract from Indonesia stimulates the whole axis of the hypothalamus, the pituitary gland, and the gonads. The stimulaton not only causes the sythesis of testosterone, but also of luteinizing hormone, follicle-stimulating hormone, and many other hormones. The syntesis of the neurotransmitter dopamine is also enhanced.

Trial participants are encouraged to keep a diary and write down episodes of sexual agitation. They could be discussed with the trial coordinator. Sexual fantasies are the clearest indication of an activated axis of the hypothalamus, the pituitary gland, and the testes. Participants should be aware that Tongkat Ali is not a medication for blood vessels. Erections of the penis are caused by changes in blood vessels, not hormonal changes. Trial participants who feel sexually aroused but suffer from vascular insufficiency could use low doses of a phosphodiesterase inhibitor together with the 1:200 Tongkat Ali Extract from Indonesia.

Ideal trial participants should be dedicated to the task of improving their libido and their sexual satisfaction.

The following dosages are recommended:

The dosage for the first day should just be one capsule. The dosage of the first day is not for a therapeutic effect. The dosage of one capsule on the first day is a test to make sure that a trial participant is not allergic to Tongkat Ali.

Tongkatali.org has been selling 1:200 Tongkat Ali Extract from Indonesia for more than 20 years. Until now, we have never met a person who is allergic to Tongkat Ali.

Allergies are unpredictable reactions of a person's immune systems. Some people are allergic to the strangest substances. There even are people who are allergic to water.

Standard dosages of Tongkat Ali would be 4 to 16 capsules a day. For adult males, it is hard to reach an overdose on Tongkat Ali. Our 1:200 Tongkat Ali Extract from Indonesia is an endocrine effector, with little or no influence on cardiovascular parameters.

Contrary to Tongkat Ali, phosphodiesterase inhibitors and similar erection medication dilate blood vessels. Erections are not caused by hormones. Erections are the result of the widening of blood vessels in the male genitals.

Erections can be caused by tactile stimulation or many physiological processes. Many of these processes are not related to sexual events. Men often have nighttime or morning erections, out of any sexual context. In many men, even plain sleepiness can cause an enlargement of the penis. Why? It happens because sleepiness is often accompanied by a drop of blood pressure, parallel to a widening of blood vessels.

Phosphodiesterase inhibitors like Viagra can cause a pronounced drop of blood pressure. This can be dangerous to the cardiovascular system. There could be a stroke, preluded by a severe headache. One clearly can overdose on Viagra, and death from phosphodiesterase inhibitors regularly are in the news.

Tongkat ali is largely neutral on the cardiovascular system. This makes Tongkat Ali less dangerous even when dosages are generous.

If Tongkat Ali raises testosterone levels, what would one feel? Higher testosterone levels, even from testosterone injections, don't makw people feel like they would have taken a drug. The most likely effect of high testosterone would be occasional sexual stimulation based on what people think or what they remember.

Trial participants should take a diary in which they make a note every time they have sexual thoughts. Previous clinical trials suggest that this will happen more often, compared to before the consumption of 1:200 Tongkat Ali Extract from Indonesia.

If sexual fantasies lead to intercourse, most men in midlife and beyond will still benefit from phosphodiesterase inhibitors like Viagra for purely physiological processes. The task of 1:200 Tongkat Ali Extract from Indonesia would be to add mental components to sexual intercourse, such as sexual desire and a sense of sexual satisfaction. These components are usually lacking with phosphodiesterase inhibitors.

Men and women who are interested to participate in our new clinical trial should write to us and attach a test results for bound and unbound testosterone.

Trial participants are entitled to receive 1:200 Tongkat Ali Extract from Indonesia at a discount of 50 percent.

Trial participants should have their blood levels tested for bound and unbound testosterone again after the consumption of the first 400 capsules of 1:200 Tongkat Ali Extract from Indonesia. The second lab report should again be sent to Tongkatali.org as email attachment.

Our experience with 1:200 Tongkat Ali Extract from Indonesia indicates that most men and women will achieve a substantial increase in testosterone. However, for any medication, there are people who do not respond. Our current clinical trial is designed to switch people who do not react on Tongkat Ali to combinations that also involve Butea Superba and Mucuna Pruriens.


Mucuna pruriens extract is about the only supplement for sexual desire


By Serge Kreutz


Neurons (nerve cells) in the human brain, mostly an area in the midbrain called substantia nigra, convert L-dopa into the neurotransmitter dopamine. Dopamine regulates motivation, including sexual motivation, and also movement.

Traditionally, a lack of sexual motivation has not been considered a clinical condition. The medical attitude has always been: no libido, no problem.

But when movement is impaired for a lack of dopamine as evident in Parkinson’s disease, this is recognized as a condition that warants medication.

The treatment of first choice for Parkinsons disease is L-dopa. The human body itself manufactures L-dopa via the amino acid L-tyrosine. The enzyme tyrosine hydroxylase does this job. This L-dopa then passes the blood-brain barrier where the another enzyme, aromatic L-amino acid decarboxylase (AADC) converts L-dopa into dopamine. This second transformation is impeded in Parkinson’s disease when decarboxylating neurons die off and are reduced in numbers. By supplying exogenous L-dopa, remaining neurons produce more dopamine. Being a biochemical which the body synthesizes on its own throughout life, the pharmaceutical industry has a hard time to patent it and make exorbitant profits.

Furthermore, L-dopa is also used in the biology of plants, most prominently in mucuna pruriens (velvet beans, cowitch... cow-itch, not co-witch).

Nevertheless, mostly two huge chemicals companies, Monsanto and Ajinomoto, have deviced methods for the industrial production of L-dopa in chemical reactions and by microbial fermentation. However, some people have voiced concerns regarding toxicity associated with these methods of industrial production, and many professionals consider the L-dopa in mucuna pruriens superior.

But pharmaceutical companies are in the business of making money, and to do so, they push their industrial products. Therefore, it's mostly the L-dopa of chemicals corporations that is used in the treatment of Parkinson’s disease.

Apart from the straight use of L-dopa, there are a few other options for increasing dopamine tone in parts of the brain. Dopamine receptor agonists are one such option. Like dopamine itself, these dopamine agonists dock on dopamine receptors (the pharmacological term is: they are ligands for dopamine receptors) and have an effect similar to dopamine. But dopamine agonists never feel as natural as plain L-dopa, and there is a plethora of definitely unwanted side effects.

But for pharmaceutical corporations, a significant aspect of dopamine agonists is that they can be patented, and can turn a profit. Thus dopamine agonists tend to be in the media much more than straight L-dopa and its most common plant source, mucuna pruriens (velvet beans).

For the pharmaceutical industry, Pfizer's Viagra was an eye-opener. It proved that enormous profits could be generated with patented sexual enhancement drugs.

Apomorphine is a case in point. Pharmaceutical companies (Tap Pharmaceuticals, Abbot Laboratories) tried to jump on the Viagra bandwagon with their own patent.

In 2001, they launched Uprima in Europe. Even back then, Uprima wasn't really a new drug. It's active ingredient, apomorphine, has been around for many years as an emetic mostly in veterinary practice. Yes, an emetic, a drug that quickly induces vomiting in dogs and other domestic animals that have swallowed some poisons.

An nausea-causing effect that is worse than the nausea-causing effect of straight L-dopa and mucuna pruriens is the most common side effect of dopaminergics.

While apomorphine has been tried as a pleasure drug, this is about all it has in common with its more famous colleague in name, morphine. Sure, apomorphine is produced from morphine. But its pharmacological effects are completely different. Morphine is a sedative agent, while apomorphine is a stimulant.

Apomorphine is a typical dopamine agonist, which accounts for its, albeit limited, usefulness in the management of Parkinson’s disease.

Apomorphine is a D1 receptor-specific dopamine agonist that makes it different from mostly ergot-derived dopamine agonists, which usually target D2 dopamine receptors, e.g. pergolide and bromocriptine. D3 and D4 dopamine receptors are less often targeted in the treatment of Parkinson’s.

It has long been documented that most Parkinson’s medications have sexuality-enhancing side effects. I was personally using mucuna pruriens and Parkinson’s medications for sexual enhancement long before Uprima was launched. I gained the most experience with L-dopa and mucuna pruriens, but I have also tested Parlodel (bromocriptine), Dopergine (lisuride), Cabergoline (brand name: Dostinex), Mirapex (pramipexole), and deprenyl.

It has to be noted that the sexuality-enhancing side effects hold true for many but not all dopamine-enhancing Parkinson’s medications. Whether or not a dopamine agonist enhances sexual functions seems to depend primarily on the dopamine receptor and sub-receptor sites it targets.

Unlike sildenafil citrate, L-dopa, mucuna pruriens, and dopamine agonists, whether Uprima or Dostinex), exert their pro-sexual effect not upon the erectile organ but upon the brain. They provoke erections not by improving the plumbing of male sexual function (i.e. blood supply to the penis), but by interfering with the wiring necessary for arousal, pleasure, and climax.

That sildenafil citrate only affects the plumbing, puts limits to its potential as a lifestyle drug. Sildenafil citrate will add little for men whose plumbing doesn’t leak, and apart from that, sildenafil also causes headache in many users, and comes with a risk of stroke. On the other hand, a good shot of additional desire is a welcome life enhancement for many people with whom there is nothing wrong physically but who just feel bored with their everyday life. For them, L-dopa, mucuna pruriens, and, on a more limited scale, dopamine agonists can be a real enrichment, and even a medication that saves their marriages.

Dosage for a pro-sexual effect is easier to determine for L-dopa and mucuna pruriens than for dopamine agonists. This is the case because a dosage that is too high will inevitably result in bad nausea. This nausea can be so bad that the last thing one fancies is sex. This particularly is a problem with apomorphine.

One of the advantages of straight L-dopa and mucuna pruriens is the fewer side effects.

Uprima has never been approved for marketing in the US, and it has meanwhile been withdrawn from Europe as a treatment for sexual dysfunction (it is still around as a veterinary emetic). The point is, apomorphine is inferior to mucuna pruriens on all counts, except profitability for pharmaceutical companies.


Read about marijuana and tongkat ali extract for meaningful sex

or

Read a tell-it-all about Indonesian 1:200 extract (and a psychopath on the prowl for killings)



Sumatra Pasak Bumi's Indonesian 1:200 tongkat ali extract more than doubled testosterone


Sumatra Pasak Bumi (main website tongkatali.org) has been selling its 1:200 Indonesian tongkat ali extract for testosterone increase for more than 20 years. We have done human studies (see below). Only 1:200 tongkat ali extract from us is genuine. All other so-called 1:200 tongkat ali extracts are simply fake.

Furthermore, only our 1:200 tongkat ali extract has undergone all the extensive testing to obtain a license as medicinal herb (Nomor Depkes) from the Indonesian government.

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Nomor Depkes


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Safety lab report

While most users can increase their testosterone levels by 30 to 50 percent through regular cycles of Sumatra Pasak Bumis 1:200 tongkat ali extract, some can achieve an increase of more than 100 percent. Here we publish several lab reports from men who achieved such a result, among them a 72-year old whose levels were raised to 224 percent above baseline.

The first lab report below shows two results, the first from January 31, 2005, before tongkat ali therapy was initiated, and the second from April 28, 2005, after three months of tongkat ali therapy. The patient was 72 years old at the time of the tests.

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Another trial participant more than tripled his levels of free testosterone.

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The following lab tests are from an athlete who used tongkat ali to raise his testosterone levels to 150 percent of normal physiological range (a level that substantially enhances muscle anabolism).

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The following test results are typical, and the effect the average user can expect. The trial participant was able to increase overall testosterone from 398 ng / dl to 477 ng / dl (a rise to 120 percent) and free testosterone from 52 pg / ml to 88 pg / ml (a rise to 169 percent), and to decrease relationships hormone binding globulin from 66 nmol / L to 43 nmol / L (a drop to 65 percent). The most dramatic change tends to be for free testosterone, which is the testosterone that really counts, because for both libido and muscle building, only free testosterone exerts an effect.

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References:

Abdul Shukor Tajul Ariff, Ima Nirwana Soelaiman, J. Pramanik, and Ahmad Nazrun Shuid, Effects of Eurycoma longifolia on Testosterone Level and Bone Structure in an Aged Orchidectomised Rat Model, Hindawi Publishing Corporation, Evidence-Based Complementary and Alternative Medicine Volume 2012 (2012), Article ID 818072, 7 pages.

Mahfoudh Abdulghani, Abas Hj Hussin, Siti Amrah Sulaiman, Kit Lam Chan, The ameliorative effects of Eurycoma longifolia Jack on testosterone-induced reproductive disorders in female rats, Science Direct, Reproductive Biology Volume 12, Issue 2, July 2012, Pages 247-255.

Catherine Ulbricht, Julie Conquer, Kelly Flanagan, Richard Isaac, Erica Rusie, and Regina C. Windsor, An Evidence-Based Systematic Review of Tongkat Ali (Eurycoma longifolia) by the Natural Standard Research Collaboration, Informa Healthcare,Journal of Dietary Supplements Vol. 10, No. 1 , Pages 54-83 March 2013.

Mahanem Mat Noor, Abu Hassan Shaari Mohd Nor, Lukman Che Hassan, The Effect of Eurycoma Longifolia Jack (Tongkat ali) on Relationships Behavior and Sperm Quality in Rats, Malaysian Journal of Pharmaceutical Sciences, vol.2, No.1, pp 53-60 (2004).



Tongkatali.org's Depressed for a reason


By Serge Kreutz


Optimal relationships experience, followed by a comfortable death, is the only sensible concept in life.

We do not live to please a specific god, or for the sake of our children, and there is no meaning in an, however identified, common good. The only perspective that makes philosophical sense is that we live to please ourselves, and orgasms are the ultimate pleasure.

There are a good number of aspects that play a role in orgasms, and their quality. Orgasms aren’t alike. Men can produce ejaculate as the result of laboring their relationships organs, almost unaccompanied by relationships fantasies or relationships pleasure. They also can ejaculate almost involuntarily, purely as a result of psychological, not physiological stimulation. There is no doubt that the second kind of orgasms provides a much higher level of satisfaction.

In accordance with the materialistic principles of science, psychological aspects have their physiological equivalents. Jealousy, for example, is an emotion, but it also is a biochemical process. Nevertheless, I sort jealousy under psychological aspects because it has a mental expression. The health of my cardiovascular system, a precondition for good erections, does not have a primary mental expression… but nevertheless greatly influences the quality of my orgasms.

I am sure that the solution to the problem of loss of excitement in orgasms will first be pharmacological, then surgical, and finally genetical. It will not be psychological, and even less philosophical. When overcoming the loss of excitement in orgasms will be as easy as stopping by a pharmacy, there will no longer be any need for treatises as the one you are currently reading. Such essays will be as unnecessary as sessions with a psychotherapist for the purpose of overcoming depression. Go and buy yourself some Prozac.

The loss of the orgasm quality is physiological. Our brains and testes no longer produce the right mix of hormones, neurotransmitters, prostaglandins, peptides, and whatever else is of relevance to afford us the ultimate bliss.

Medical science so far does not concern itself much with orgasm quality, but there already are prescription pharmaceuticals, dopaminergics, that somehow improve orgasms. These drugs are used in the treatment of Parkinson’s disease. However, in people not afflicted with Parkinson’s, they tend to cause nausea. Worst in this respect is lisuride.

While the nausea may be bearable for some people more than for others, these Parkinson’s medications are prescription drugs all the same for everyone. Some of them are also extremely expensive.

Butea superba, a Thai herbal, is probably the only pharmacological agent that improves orgasm quality without side effects.

Butea superba has a unique double mode of action by enhancing testosterone synthesis and inhibiting phosphodiesterase at the same time.

Phosphodiesterase inhibition is the route of action of prescription drugs for erectile dysfunction.

But butea superba doesn’t feel like these prescription drugs. Butea superba facilitates erections more naturally because they happen in tandem with heightened libido.

And then, more specifically, butea superba extends the time frame of the pre-orgasmic plateau.

For most men, the pre-orgasmic plateau is just 2 or 3 seconds, and younger men often don’t know how to enjoy it.

The pre-orgasmic plateau is the moment when male ejaculation becomes certain, regardless of whether penetrative thrusting is continued or not. Physiologically, it is the time when sperm and the fluids of the seminal vesicles accumulate at the base of the urethra for expulsion.

This phase of the orgasm is already highly pleasurable, even though younger men are hardly aware of it. Older men more often can enjoy this phase, and they stop penetration, and let it come all by itself.

Butea superba can extend this plateau phase, and give it a duration of 5 to 10 seconds, which feels like an eternity of the most exquisite pleasure.

Because the directors of porn movies usually demand that ejaculation happens in front of the lens, rather than inside the female body, studs often supplement with butea superba. Not only does butea superba help them to stay focused on their assignment of the day in spite of unromantic onlookers; butea superba also allows them more time to withdraw from the woman and position their vital organ before the camera before shooting their loads.


Butea superba in sports doping


By Serge Kreutz



Doping in sports has a bad name. But what is doping, and what is not?

Lance Armstrong had testicular cancer that warranted treatment with certain medications. Some of these medications can be used to enhance performance in sports.

Actually, anything that puts an athlete at optimal health is performance enhancing.

Athletes treat infections with antibiotics. The antibiotics certainly help performance by eliminating performance-limiting obstacles.

Athletes of any specialty are typically very health-conscious. Their diets often include vitamins and supplements. And athletes use them for performance-enhancing properties.

What is doping, what is not? The boundary is hard to draw.

Athletes consult physicians on how to bring their bodies into optimal states to win in sports competitions. To ask physicians is an obvious choice. Physicians know human physiology. Physicians prescribe medications to improve or tilt homeostasis specifically for certain sports events. But what is improvement of health, what is doping?

High altitude training enhances the body’s ability to utilize oxygen. Some sports, like long-distance running, is dominated by athletes from high-altitude countries.

And in weightlifting, a stunted growth is often of advantage, and people from ethnicities where stunted growth is more common, tend to dominate.

Or look at Olympic swimmers, especially the women’s competition. Some participants who do well look like having a past of anabolics use. To say the least.

Butea superba enhances health.? And not just that. It enhances hormonal health. But unlike doping with anabolic steroids, butea superba activates the whole hypothalamic-pituitary-gonadal axis.?

The effect is something like genetically derived high testosterone. And that’s beneficial for athletic performance and overall health, and in the mating arena, too.

Will butea superba be detected in anti-doping tests that target steroids use? Probably not. Anti-doping tests typically measure ratios of certain hormones related to the hypothalamic-pituitary-gonadal axis.

In a 2008 scientific study named “The effect of butea superba roxb., a Thai traditional medicine, on endogenous steroid levels in males”, conducted by the Department of Pharmacology of the prestigious Thai Mahidol University in cooperation with the Thai National Anti-Doping Centre, it was found that the levels of endogenous steroids were raised, but their ratios were unchanged. Here quotes from the abstact of the study:

“After oral administration of ground Butea superba Roxb. root and tuber, twenty-four hour urines were collected and analysed for endogenous steroids by GC/MS. The amount of LH was also determined in the same sample by fluoroimmunoassay… Elevations of several endogenous steroids were observed after short term administration of Butea superba Roxb. Among these are androstendione, testosterone, estradiol and estriol. The concentration of the glucocorticosteroids, cortisol and tetrahydrocortisol, were unaffected. Slight increase in urinary LH was observed. No significant change in T/DHT, androsterone/etiocholanolone and T/estradiol ratios were observed.”

Butea superba is well established in sports communities that enhance results by pharmacological means. Among scientifically-oriented, steroids-using bodybuilders, butea superba is commonly added to on-cycle stacks to avoid a complete shutdown of the hypothalamic-pituitary-gonadal axis when using heavy calibers like Trenbolone, and for the purpose of maintaining libido when on “juice”.

Butea superba is also well-established for post-steroid cycle recovery, when there is a definite need to stimulate the hypothalamic-pituitary-gonadal axis to get back to normal hormonal profiles.

However, in sports that qualify for Olympic competition, the use of butea superba has so far been limited. Sure, butea superba isn’t in the a league with performance enhancers such as EPO or standard testosterone.

But a performance improvement of just 5 or 10 percent can make a difference between a medal or no medal. Even 2 percent can.

And butea superba’s function and classification as health supplement, not as drug, is of great advantage.

Butea superba doesn’t cause doping-positive results because hormonal profiles stays in natural delimitations, with natural hormonal ratios.


References:

Chaiyasit, K., Wiwnaitkit, V. (2012) Hyperandrogenemia due to ingestion of Butea superba. Indian Journal of Endocrinology and Metabolism Volume 16 Issue 3 Pages: 485–486 Tongkatali.org Bibliography

Cherdshewasart, W., Nimsakul, N. (2003) Clinical trial of Butea superba, an alternative herbal treatment for erectile dysfunction. Asian Journal Andrology Volume 5 Pages: 243-246 Retrieved from: Tongkatali.org Bibliography

Eumkeb, G., Naknarong, W., Sirichaiwetchakoon, K. (2013) The effects of Red Kwao Krue (Butea Superba Roxb.) extract on sperm quality and testosterone level in mice. Thai Journal of Pharmaceutical Sciences Volume 38 (Suppl.) Retrieved from: Tongkatali.org Bibliography

Malaivijitnond, S., Ketsuwan, A., Watanabe, G., Taya, K., Cherdshewasart, W. (2010) Luteinizing hormone reduction by the male potency herb, Butea superba Roxb. Brazilian Journal of Medical and Biological Research Volume 43 Number 9 Tongkatali.org Bibliography

Malviya, N., Jain, S., Gupta, V.B., Vyas, S. (2011) Recent studies on aphrodisiac herbs for the management of male relationships dysfunction - a review. Acta Poloniae Pharmaceutica Volume 68 No. 1 Pages:3-8, Tongkatali.org Bibliography

Tocharus, C., Jeenapongsaa, R., Teakthonga, T., Smitasirib, Y. (2005) Effects of Long-term Treatment of Butea superba on Sperm Motility and Concentration. Naresuan University Journal Volume 13 Issue 2 Pages: 11-17

Tocharus, C., Sooksaen, P., Shimbhu, D., Tocharus, J. (2011) Butea superba (Roxb.) improves penile erection in diabetic rats. Andrologia, Volume 44, Issue 1 Pages: 728-733 Tongkatali.org Bibliography



Tongkatali.org - Black ginger is an amazing herb


By Serge Kreutz


Black ginger is an amazing herb. It has so many health benefits. It's protective against diabetes and obesity, it's a true aphrodisiac, making relationships much better. And it even can revert cognitive impairment (dementia).

This is all scientifically proven. Just check the Medline database of the National Institutes of Health of the US government. There you can find many scientific studies. But you will have to use the scientific Latin name for searching Medline. The scientific name of the plant is Kaempferia parviflora.

This plant has so many names. It is also called black galingale or galangale. And then there is the name krachai dam. That is the official transcription of the Thai characters. But in English, krachai dam is also spelled krachai dum, or kra chai dam, or grachai dam or dum. The names of this plant are really quite confusing. We stick mostly to black ginger.

Most of the benefits of black ginger are related to the plant's content of phosphodiesterase inhibitors. Yeah, phosphodiesterase inhibitors. That's the stuff medications for erectile dysfunction are made of.

Phosphodiesterase inhibitors stop phosphodiesterase from breaking down cGMP and cAMP. The last two prevent the constriction of blood vessels.

There are so many diseases in modern humans that are all related to the constriction of blood vessels: high blood pressure, heart attack, stroke, erectile dysfunction, diabetes, and even premature aging.

Keep the blood flowing! This is an essential physiological function. And this is where black ginger is of help. Consume it generously, every day of the year. Make it a tea. Add it to coffee. Put it in any sauce. Even to backed sweets. While the flavor of black ginger isn't as strong as that of yellow ginger, it never destroys the flavor of the dish where you add it. Using 100 grams per day in various forms won't have ill effects.

And if you are involved in competitive sports, you have an extra reason to use black ginger. Numerous studies have shown that it increases physical strength and endurance. Check Medline.

The plant is always used in camps of Thai kick boxers. Obviously, more blood flow to muscles increases strength. And the plant has even been named scientifically for this application, the use in kick boxing. Kaempfer is the German word for fighter, and kaempferia is the Latinized version. So, in common language, Kaempferia parviflora is just Fighter's parviflora.

Keep the blood flowing! What we mean is: inside your blood vessels. Not: from the wounds of your enemies.

Keep the blood flowing inside your arteries and veins. For a better oxygen supply to your muscles and heart, and, of course, for better erections.



Tongkat Ali (Eurycoma longifolia Jack): A review on its ethnobotany and pharmacological importance

Rajeev Bhat, A.A.Karim

Abstract

Eurycoma longifolia Jack is an herbal medicinal plant of South-East Asian origin, popularly recognized as ‘Tongkat Ali.’ The plant parts have been traditionally used for its antimalarial, aphrodisiac, anti-diabetic, antimicrobial and anti-pyretic activities, which have also been proved scientifically. The plant parts are rich in various bioactive compounds (like eurycomaoside, eurycolactone, eurycomalactone, eurycomanone, and pasakbumin-B) among which the alkaloids and quassinoids form a major portion. Even though toxicity and safety evaluation studies have been pursued, still a major gap exists in providing scientific base for commercial utilization and clearance of the Tongkat Ali products with regard to consumer's safety. The present review aims at reviewing the research works undertaken till date, on this plant in order to provide sufficient baseline information for future works and for commercial exploitation.

https://doi.org/10.1016/j.fitote.2010.04.006




PT Sumatra Pasak Bumi
7th floor, Forum Nine
Jl. Imam Bonjol No.9
Petisah Tengah
Medan Petisah
Medan City
North Sumatra 20236
Indonesia
Tel: +62-813 800 800 20